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Posts by Gay PEDOPHILE terrorist serial killer I jack off to dead babies


  1. THROW IT IN A WORMHOLE

    CUZ IN THE END WERE ALL ALONE

    DO YOU BELIEVE IN GOD

    WHO DO YOU THINK MADE THE INTERNET

    I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If tceptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I

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  2. THROW IT IN A WORMHOLE

    CUZ IN THE END WERE ALL ALONE

    DO YOU BELIEVE IN GOD

    WHO DO YOU THINK MADE THE INTERNET

    I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If tceptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I

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    In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because

  3. THROW IT IN A WORMHOLE

    CUZ IN THE END WERE ALL ALONE

    DO YOU BELIEVE IN GOD

    WHO DO YOU THINK MADE THE INTERNET

    I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't proddsolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because

  4. THROW IT IN A WORMHOLE

    CUZ IN THE END WERE ALL ALONE

    DO YOU BELIEVE IN GOD

    WHO DO YOU THINK MADE THE INTERNET

    I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because Candiria (which is a shitty hardcore band) made an album called that and the album cover is quite similar to the hallucination. I also masturbate without any porn half the time because I just visualize the sex. I have it pretty strong. It's not unbearable static like some people get it, but I can see patterns appearing in my vision, especially with my eyes closed, almost all of the time, and I can consciously manipulate my hallucinations so that I feel like I'm tripping even when I''m sober i.e. the psuedohallucinations are interactive and controlled. I'll give an example of the visual phenomena I am experiencing at the moment. Looking around, everything is in a very, very mild multicolored static which is only noticeable when you purposely try to look at it. When I look into the distance I can make the fuzz take on somewhat geometric forms. I can auto-kick the derealization of bundy into gear, and I can move things in my field of vision slightly by staring at them. With closed eyes, I'm getting a vague image of an ovary or a penis in blue static in front of the black behind the eyelids. Do meditative techniques increase visual hallucinations? In fact, I'm really curious in general neurotransmitter manipulation by conscious though. Like, would it be possible to achieve significantly altered states of consciousness with only endogenous neurochemicals? If there's a free acid trip burrowed in my brain somewhere hell yeah I'd take it. I wonder if HPPD is a form of receptor rebound.. Like, the 5ht2c receptors get antagonized leading to a particular secondary messenger pathway when psychedelics are ingested, and then this antagonism distorts perception and feedback inhibition in optic nerves, leading to an exaggeration of certain elements, an inconsistent recurrence of specific optic networks resulting in visual pattern hallucinations. NMDA antagonists instead of working on feedback inhibition, which is the reduction of neural activity after a specific metric of signals have been sent…work by inducing sensory shutdown, with NMDA receptors turning off less essential neural networks and leaving a more basic brain which in turn has to use its energy to "fill in the blanks" induced by the shut down neural circuits, so the more essential neural circuits induce hallucinations so that your brain isn't processing absolutely nothing. Anticholinergics I'm not so sure about, I know choline converts to acetylcholine and this is an important neurotransmitter for muscle contractions as well as learning…so I guess a lack of choline causes a sensory shutdown which isn't a true deactivation of neural networks, but all the neural networks you typically have become weak and aperceptive, just like your muscles become weak. Then coupled with the dysphoria, when your brain tries to fill in the blanks, you get spiders. Not sure about cannabinoid 1 receptors either but I think the psychedelic qualities might have to do with the induction of neurogenesis. The only other receptors I can think of which may be implicated in hallucinations and schizophrenia is the kappa opiod receptors. I've never gotten high on salvia before so I can't really make any assumptions on what it's general properties or mechanism of action for hallucinations is. Guided meditation alongside foods that increase specific neurotransmitter levels or act as weak versions of these drugs (especially certain herbal teas) might have some promise for those dedicated to a spiritual experience. I consider the little temple I see in the back of my eyelids when I close my eyes to be the home of my consciousness, looked at from an alternate entity's perspective. I call it the Temple Of The Sickness, because

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